« Previous
Next »
The Journal of Heart and Lung Transplantation
Volume 29, Issue 5
, Pages 554-561
, May 2010
Duration of left ventricular assist device support: Effects on abnormal calcium cycling and functional recovery in the failing human heart
References
- Abnormal intracellular calcium handling in myocardium from patients with end-stage heart failure. Circ Res. 1987;61:70–76
- Alterations of contractile function and excitation contraction coupling in dilated cardiomyopathy. Circ Res. 1992;70:1225–1232
- Decreased catecholamine sensitivity and beta-adrenergic-receptor density in failing human hearts. N Engl J Med. 1982;307:205–211
- Diminished post-rest potentiation of contractile force in human dilated cardiomyopathy (Functional evidence for alterations in intracellular Ca2+ handling). J Clin Invest. 1996;98:764–776
- . LVAD-induced reverse remodeling: basic and clinical implications for myocardial recovery. J Card Fail. 2006;12:227–239
- Improved left ventricular function after chronic left ventricular unloading. Ann Thorac Surg. 1996;62:675–682
- Cardiac improvement during mechanical circulatory support: a prospective multicenter study of the LVAD Working Group. Circulation. 2007;115:2497–2505
- Effect of the implantable left ventricular assist device on neuroendocrine activation in heart failure. Circulation. 1995;92:II-191-5
- Circulatory resuscitation with left ventricular assist device support reduces interleukins 6 and 8 levels. Ann Thorac Surg. 1997;63:971–974
- Regression of cellular hypertrophy after left ventricular assist device support. Circulation. 1998;98:656–662
- Structural and left ventricular histological changes after implantable LVAD insertion. Ann Thorac Surg. 1995;59:609–613
- Cytoskeletal structure and recovery in single human cardiac myocytes. J Heart Lung Transplant. 2004;23:954–963
- Altered myocardial phenotype after mechanical support in human beings with advanced cardiomyopathy. J Heart Lung Transplant. 1997;16:765–773
- Mechanical unloading leads to echocardiographic, electrocardiographic, neurohormonal, and histologic recovery. J Heart Lung Transplant. 2006;25:7–15
- Assist devices fail to reverse patterns of fetal gene expression despite β-blockers. J Heart Lung Transplant. 2007;26:1170–1176
- Myocyte recovery after mechanical circulatory support in humans with end-stage heart failure. Circulation. 1998;97:2316–2322
- Mechanical unloading restores beta-adrenergic responsiveness and reverses receptor downregulation in the failing human heart. Circulation. 2001;104:881–886
- Weaning from mechanical cardiac support in patients with idiopathic dilated cardiomyopathy. Circulation. 1997;96:542–549
- Low incidence of myocardial recovery after left ventricular assist device implantation in patients with chronic heart failure. Circulation. 1998;98:2383–2389
- Left ventricular assist device and drug therapy for the reversal of heart failure. N Engl J Med. 2006;355:1873–1884
- Long-term use of a left ventricular assist device for end-stage heart failure. N Engl J Med. 2001;345:1435–1443
- Outcomes of left ventricular assist device implantation as destination therapy in the post-REMATCH era: implications for patient selection. Circulation. 2007;116:497–505
- Prediction of cardiac stability after weaning from left ventricular assist devices in patients with idiopathic dilated cardiomyopathy. Circulation. 2008;118(suppl 1):S94–S105
- Which factors predict the recovery of natural heart function after insertion of a left ventricular assist system?. J Heart Lung Transplant. 2008;27:869–874
- Changes in sarcomeric and non-sarcomeric cytoskeletal proteins and focal adhesion molecules during clinical myocardial recovery after left ventricular assist device support. J Heart Lung Transplant. 2007;26:230–235
- Role of intracellular calcium handling in force-interval relationships of human ventricular myocardium. J Clin Invest. 1990;85:1599–1613
- Alterations in intracellular calcium handling associated with the inverse force–frequency relation in human dilated cardiomyopathy. Circulation. 1995;92:1169–1178
- Design of a phase 1/2 trial of intracoronary administration of AAV1/SERCA2a in patients with heart failure. J Card Fail. 2008;14:355–367
- Chronic unloading by left ventricular assist device reverses contractile dysfunction and alters gene expression in end-stage heart failure. Circulation. 2000;102:2713–2719
- Sarcomeric genes involved in reverse remodeling of the heart during left ventricular assist device support. J Heart Lung Transplant. 2005;24:73–80
- Comparison of right and left ventricular responses to left ventricular assist device support in patients with severe heart failure (A primary role of mechanical unloading underlying reverse remodeling). Circulation. 2001;104:670–675
- Altered myocardial Ca2+ cycling after left ventricular device support in the failing human heart. J Am Coll Cardiol. 2004;44:837–845
- Time course of reverse remodeling of the left ventricle during support with a left ventricular assist device. J Thorac Cardiovasc Surg. 2001;121:902–908
- Inotropic effects of angiotensin II on human cardiac muscle in vitro. Circulation. 1990;82:1973–1984
- Beta-adrenergic receptors and calcium cycling proteins in non-failing, hypertrophied, and failing human hearts: transition from hypertrophy to failure. J Mol Cell Cardiol. 2001;33:1283–1295
- . The relative phospholamban and SERCA2 ratio: a critical determinant of myocardial contractility. Bas Res Cardiol. 1997;92:17–24
- Altered myocardial force-frequency relation in human heart failure. Circulation. 1992;85:1743–1750
- Relation between myocardial function and expression of sarcoplasmic reticulum Ca(2+)-ATPase in failing and non-failing human myocardium. Circ Res. 1994;75:434–442
- Reduced contraction and altered frequency response of isolated ventricular myocytes from patients with heart failure. Circulation. 1995;92:2540–2549
- Mechanical support in dilated cardiomyopathy: signs of early left ventricular recovery. Ann Thorac Surg. 1997;64:1301–1308
- Alterations in Ca2+ cycling proteins and Gαq signaling after left ventricular device support in failing human hearts. Cardiovasc Res. 2000;45:883–888
- Cardiac contractility modulation electrical signals improve myocardial gene expression in patients with heart failure. J Am Coll Cardiol. 2008;51:1784–1789
- Myocardial gene expression in heart failure patients treated with cardiac resynchronization therapy responders versus nonresponders. J Am Coll Cardiol. 2008;51:129–136
- Evidence for functional relevance of an enhanced expression of the Na+–Ca2+ exchanger in failing human myocardium. Circulation. 1996;94:992–1002
- PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts. Cell. 2000;101:365–376
- Partial loading of the left ventricle during mechanical assist device support is associated with improved myocardial function, blood flow and metabolism, and increased exercise capacity. J Heart Lung Transplant. 2002;21:446–454
Current affiliation for M.L.O: Department of Molecular Physiology and Biophysics, University of Houston, Houston, Texas.
PII: S1053-2498(09)00848-1
doi: 10.1016/j.healun.2009.10.015
© 2010 International Society for Heart and Lung Transplantation. Published by Elsevier Inc. All rights reserved.
« Previous
Next »
The Journal of Heart and Lung Transplantation
Volume 29, Issue 5
, Pages 554-561
, May 2010
